A locally-built, LLM-digested index of recent arXiv papers in quant finance, geometry/topology, and statistical ML — keyword search served straight from SQLite on this machine.
Develops geometric causal models for causal inference from dependent data.
problem Causal inference from structured, dependent data (e.g., spatial, network, molecular).
method Geometric causal models (GCMs) exploiting symmetries of data generating process, combining group theory, ergodic theory, and Bayesian inference.
result Establishes identification and estimation of causal effects from dependent data.
Study identifies and estimates treatment effect heterogeneity within principal stratification subpopulations.
problem Causal inference with intermediate outcomes and treatment effect heterogeneity.
method Proposes a novel doubly cross-fit doubly robust machine learner to efficiently learn conditional principal causal effects under principal ignorability.
result Demonstrates informative patterns of treatment effect heterogeneity within the always-survivor subpopulation in an acute lung injury trial.
Causal inference from observational data is hard due to discontinuous causal effects.
problem Causal inference from observational data is hard due to discontinuous causal effects.
method The problem is tackled by showing that many standard point estimates can be read as point summaries of multimodal distributions over the space of structural causal models.
result Many standard point estimates can be discontinuous summaries, while explicit posterior means and medians are continuous.
Two environments are enough to infer causal graphs and counterfactuals.
problem Inferring causal relations from multiple environments, especially for nonlinear mechanisms.
method Using structural causal models and the invariance principle, the study shows that only two auxiliary environments are sufficient for causal graph inference and counterfactual inference.
result Two auxiliary environments are sufficient for identifying causal graphs and counterfactuals.
Develops a new approach for algorithmic recourse in AI systems.
problem Tackles the problem of providing recommendations for reversing negative AI decisions.
method Introduces a causal framework that models recourse as a process over pre- and post-intervention outcomes, allowing for partial stability and resampling of latent variables.
result Demonstrates the value of the proposed methods on real and semi-synthetic datasets.
New method detects bias in AI models that generate data.
problem Detecting bias in AI models that generate data.
method Formalized causal fairness in generative AI, derived new decomposition results, established identification conditions, and introduced efficient estimators.
result Demonstrated the value of new methodology in analyzing bias in large language models.
MOCA uses modular attention to estimate causal effects from complex data.
problem Estimating causal effects from observational data with complex, non-linear, and high-dimensional treatment and outcome mechanisms.
method MOCA is a transformer-based framework that separates treatment and outcome modeling through modular design and one-way attention mechanism, with cutting-feedback to prevent outcome influence on treatment representations.
result MOCA outperforms classical estimators and machine learning approaches across various simulated and real-world scenarios.
Generative Augmented Inference improves AI-generated data for causal inference.
problem Challenges in using AI-generated annotations for reliable causal inference.
method Generative Augmented Inference (GAI) treats AI outputs as informative features for learning true labels, flexibly modeling the relationship using nonparametric methods.
result GAI significantly reduces estimation error and improves confidence interval quality compared to human-only and PPI-based methods.
IIC decouples causal identification into two phases, significantly reducing the HTC gap in linear SEMs.
problem Determining causal effect coefficients in linear SEMs with latent confounders using the Half-Trek Criterion (HTC) leaves a gap of inconclusive causal effects.
method Iterative Identification Closure (IIC) framework that decouples causal identification into two phases: a seed function S_0 and Reduced HTC propagation.
result IIC strictly subsumes both HTC and ancestor decomposition, reducing the HTC gap by over 80% with combined seeds.
Single proxy variable helps estimate causal effects from confounders.
problem Estimating causal effects from treatment to outcome when unobserved confounders are present.
method Assumes a single, potentially multi-dimensional proxy variable of the unobserved confounder and a known mechanism generating the proxy from the confounder. Proves causal effects are identifiable under completeness assumption.
result Causal effects are identifiable under SPICE assumption.
Tests whether a treatment's effect is fully mediated by observed outcomes and identifies causal mechanisms.
problem Understanding how a treatment affects an outcome through intermediate variables.
method Proposes a test to evaluate full mediation and causal mechanism identification, extending to non-randomly assigned treatments.
result A conditionally random treatment is conditionally independent of the outcome given mediators and covariates if full mediation and causal mechanism identification hold.
CausalMix generates synthetic data with causal controls for mixed-type tables.
problem Synthetic data for causal inference with mixed-type and multimodal tabular data.
method CausalMix combines Gaussian latent priors with data-type-specific decoders for control over overlap, confounding, and treatment effect heterogeneity.
result CausalMix achieves state-of-the-art distributional metrics and stable causal control.